Whether you live in a house or apartment or simply need to ensure that your house is free from EMFs, there are a number options to limit your exposure. One of the most effective is to limit your use of electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from entering your home. Another easy way to protect your home against EMF radiation is to put up an RF shielding canopy. This is a cloth of net that contains EMF shielding and is used to block EMFs from entering a space. Another alternative is to have your home fitted with a conductive enclosure. They are referred to as Faraday cages.
emf blockers have demonstrated that the non-ionizing RF EMF can cause antiproliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to involve down-regulation the cancer stem cell. This could explain the long-term response seen in some patients with advanced HCC. But, the reason for AM RF EMF's effect in patients suffering from cancer isn't yet clear.
Aspects from AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. emf blocker were divided in three different groups. One group did not have exposure RF EMF. Another group of participants was subjected RF EMF at frequencies similar to the frequency used by humans. In the third, they were subjected to RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on tumours was assessed against the effect of RCF. The results indicated that cancers treated with HCCMF had significant shrinkage. However, the tumors treated with RCF did not show any evidence of shrinkage of the tumor.
The mechanism behind tumor-specific AM RF EMF might be based on the fact that cancer cells require Cav3*2 voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is caused through CACNA1H the protein which is responsible for the influx of Ca2+ specific to tumours. block emf suggest that CACNA1H could have more broader implications for the treatment and diagnosis of many cancers.
The tumours of the controls were never exposed to EMF from RF, and fed a standard mouse diet. The tumors in the HCCMF group were infected with Huh7 cells after they were 5 to 7 weeks old. The tumours were then euthanized in cases of excessive burden.
The tumours from the three groups also displayed different growth curves. The tumours treated with HCCMF showed a significant decrease in the size of the tumour after 8 weeks. However, the tumors which were treated by RCF didn't show signs of shrinkage. The difference was substantial. The tumours treated with RCF were able to show necrosis that is common in tumours that are exposed to RCF. There is a possibility that the necrosis was due to an absence of oxygen in larger tumours.
In conclusion, the findings show an AM-RF EMF has anticancer properties in vitro as well as in live. A number of studies have proven it is true that AM RF EMF produces measurable shrinkage of tumors in HCC patients. There is a possibility that AM RF EMF triggers these effects due to CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained impact on the development of HCC tumours in living tissue.