EMF Blocking and even EMF Shielding

· 2 min read
EMF Blocking and even EMF Shielding

If you reside in an apartment or house or simply need to ensure that your house is free from EMFs There are plenty of methods to reduce exposure. One of the easiest is to reduce the use of electronic devices. You can also turn to EMF block paint to prevent EMF radiation from entering your home. Another way to shield your house from EMF radiation is to install a RF shielding canopy. This is a cloth of net that contains EMF shielding. It's used to block EMFs from entering a room.  emf blockers for phones  is to have your home fitted with an enclosure that is conductive. These devices are called Faraday cages.

A number of studies have proven that the non-ionizing RF EMF has antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed to involve down-regulation in cancer-related stem cells.  phone emf blocker  could be the reason for the long-term responses seen in certain patients suffering from advanced HCC. However, the mechanism behind AM EMF's effects on patients with cancer is not evident.

Aspects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were examined in mice. The tumors were split in three different groups. The first group was not exposed to RF EMF. Second group members were exposed RF EMF at frequencies similar to that used in humans. The third group was exposed to the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was assessed against the effect of RCF. The results indicated that cancers treated with HCCMF showed significant shrinkage. However, the tumors treated with RCF did not show any evidence of shrinkage of the tumor.

The mechanism of tumor-specific AM RF EMF could be due to the fact that tumour cells require Cav3*2 voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is mediated through CACNA1H, a protein that is responsible for the influx of Ca2+ specific to tumours. The results suggest that CACNA1H could have wider implications in the diagnosis and treatment of various cancers.

The tumours in the controls were never exposed EMF from RF, and fed a normal diet of mice. The tumors of the HCCMF group were injected with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were then killed when they showed excessive burden.

The tumours from the three groups also displayed distinct growth curves. The tumors treated with HCCMF had a significant reduction in size of the tumor after 8 weeks. However, tumors treated with RCF did not show any reduction in size. The difference was highly significant. The tumours treated with RCF showed necrosis, which is typical when tumors are exposed to RCF. There is  emf neutralizer  that this necrosis is caused by the lack of oxygen in larger tumors.


In sum, the results indicate that AM RF EMF exhibits anticancer activity in vitro and in live. A number of studies have proven it is true that AM RF EMF produces measurable shrinkage of tumors for HCC patients. There is a possibility that AM RF EMF causes these effects due to CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained effect on the development of HCC tumors in the vivo.